Frontiers | The Promiscuous Profile of Complement Receptor 3 in Ligand Binding, Immune Modulation, and Pathophysiology
Pericyte stem cells induce Ly6G+ cell accumulation and immunotherapy resistance in pancreatic cancer | EMBO reports
Confluence Mobile - Centenary Institute Confluence Wiki
File:Fimmu-09-02733-g002.jpg - Wikimedia Commons
The TAM family receptor tyrosine kinase TYRO3 is a negative regulator of type 2 immunity | Science
FCSalyzer / Wiki / Transformation
Frontiers | Positive Allosteric Modulation of CD11b as a Novel Therapeutic Strategy Against Lung Cancer
A Clonogenic Bone Marrow Progenitor Specific for Macrophages and Dendritic Cells | Science
Neutrophil - Wikipedia
GB1275, a first-in-class CD11b modulator: rationale for immunotherapeutic combinations in solid tumors | Journal for ImmunoTherapy of Cancer
Confluence Mobile - Centenary Institute Confluence Wiki
Confluence Mobile - Centenary Institute Confluence Wiki
GB1275, a first-in-class CD11b modulator: rationale for immunotherapeutic combinations in solid tumors | Journal for ImmunoTherapy of Cancer
ATF3 coordinates serine and nucleotide metabolism to drive cell cycle progression in acute myeloid leukemia - ScienceDirect
Engineered Lactococcus lactis secreting Flt3L and OX40 ligand for in situ vaccination-based cancer immunotherapy | Nature Communications
Epithelial colonization by gut dendritic cells promotes their functional diversification - ScienceDirect
Frontiers | Positive Allosteric Modulation of CD11b as a Novel Therapeutic Strategy Against Lung Cancer
IJMS | Free Full-Text | Chronic Intermittent Hypoxia during Sleep Causes Browning of Interscapular Adipose Tissue Accompanied by Local Insulin Resistance in Mice
Trogocytosis - Wikipedia
AML (Acute Myelogenous Leukemia with maturation) (M2) | Flow Cytometry
Confluence Mobile - Centenary Institute Confluence Wiki
AMoL (Acute Monoblastic/Monocytic Leukemia) (M5) | Flow Cytometry
CD11b expression distinguishes sequential stages of peritoneal B-1 development | PNAS
Frontiers | Nrf2 Is a Central Regulator of Metabolic Reprogramming of Myeloid-Derived Suppressor Cells in Steady State and Sepsis
